PHI-base : The Pathogen - Host Interaction Database

This database contains expertly curated molecular and biological information on genes proven to affect the outcome of pathogen-host interactions. Information is also given on the target sites of some anti-infective chemistries.
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Version: 3.1 beta Last revision: 22/10/07

This site is sponsored by Rothamsted Research and The Biotechnology and Biological Sciences Research Council (BBSRC, UK) and is being developed and maintained by scientists at Rothamsted Research

Allylamines
Benzimidazoles
Carboxylic acids
Dicarboximides
Hydroxyanilides
Methyoxyacrylates
Morpholines
N-phenyl carbamates
Oxathiin carboxamides
Piperazines
Thiophanates
Triazoles

Loss of pathogenicity
Reduced virulence
Unaffected pathogenicity
Increased virulence (Hypervirulence)
Enhanced antagonism
Wild-type mutualism
Effector (plant avirulence determinant)
Resistant to chemical
Sensitive to chemical
Susceptible to chemical
Lethal

gene disruption
+ gene mutation
+ gene mutation: characterised
+ gene deletion; complementation
+ gene deletion
+ complementation
+ complementation; other evidence
+ altered gene expression: overexpression
+ altered gene expression: overexpression; complementation
gene deletion
+ altered gene expression: overexpression
+ complementation
+ complementation; biochemical evidence
+ biochemical analysis
cluster gene deletion
altered gene expression / gene regulation
+ downregulation
+ overexpression
+ silencing
+ down- and upregulation
+ complementation
biochemical analysis
+ mutation: characterised
+ mutation: characterised; complementation
functional test in host
+ direct injection
+ transient expression
mutation
+ characterised
+ characterised; complementation
complementation
sequence analysis of sensitive and resistant strains
sexual cross, sequencing of resistance conferring allele
other evidence